How lifestyle habits and stressors affect NAD+ levels
The idea of a healthy lifestyle has permeated modern culture, and for good reason. Healthy lifestyle practices help to combat the numerous stressors we all face every day—stressors that affect NAD+ levels.
How lifestyle stressors affect your cells
The biggest stressors that affect your health (and NAD+ levels)
When we refer to “lifestyle stress management” we’re looking at common lifestyle parameters that are known to deeply affect one’s health over time. They are:
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Time (age)
Ageing occurs due to the passage of time. The more time your cells spend under physiological stress, the more subject they are to wear and tear.
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Dietary distress
When you eat poorly, your NAD+ levels and the cellular processes supported by steady NAD+ production can suffer.
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Immune stress
There's a name for the ageing of your immune system: immunosenesence. As you age, protecting the health of your cells is more important than ever.
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Sleep disruption
Interrupted circadian rhythm can deplete NAD+, which decreases your cells' ability to make energy, carry out repair, and function overall.
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Overtraining
Overtraining risks injury, which requires more NAD+ to be shuttled to repair processes.
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Environmental stressors
Daily environmental factors such as smoke and pollution can trigger oxidative stress, leading to a decline in NAD+ and mitochondrial health.
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Alcohol consumption
Alcohol can trigger oxidative stress and cause free radicals, which can damage DNA. Overdoing it is unkind to your cells.
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Sedentary lifestyle
Inactivity has been linked to shortened telomeres caused by oxidative stress and free radicals, leading to NAD+ decline.
Cellular stressors deplete NAD+
All of these stressors can damage your cells over time, which can create a cycle of depleted NAD+, leading to more cellular dysfunction, leading to less NAD+, and on and on. The loop of cellular stress and NAD+ depletion can accelerate the signs of ageing over time, and is implicated in the development of a wide range of health conditions.
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